If you had been going down the rabbit hole of COVID-19 research for long enough, a few things would be astounding to you.

Fauci, can you please explain how COVID-19 is a novel virus when it has an overlap with the structure of SARS at a rate of 79%?

All coronaviruses have the telltale spike protein we have all become familiar with and MERS was less similar to SARS than COVID-19 is.

Often news reports are discussing new mutations of COVID-19.

Aren’t virus mutations expected and common.

It appears that there has not been a mutation that significantly increases the way the virus works in the body making it more deadly or dangerous.

Both SARS and COVID-19 have a spike protein that uses angiotensin-converting enzyme 2 (ACE-2) receptors to enter the respiratory tract cells, correct.

I understand COVID-19 may be more efficient at doing so, but the cell entry is highly similar.

In 2005 the NIH did a study on chloroquine that found it was effective in inhibiting and eliminating the SARS virus in vitro.

In 2003 an article in New Scientist documented that patients who died from SARS actually died of a cytokine storm. This appeared to be well known in the research community?

There are now several studies that indicate that some significant portion of the population has T-cell or long-term immunity to COVID-19 due to exposure to coronaviruses in the past.

How does this research impact public health measures and the approach to the virus.

The PCR type test looks for pieces of the COVID-19 virus RNA.

Because I actively look for information and follow the data about COVID-19, it is easy to see there is a lot of good news.

This is not a novel virus and it works in ways very similar to SARS.

The virus is fading in the sunbelt and there are very good data to believe there are large swaths of the population that will not become ill with it.